For low-carbohydrate diets, a public health approach has focused on the replacement of carbohydrates with unsaturated fats. However, little research exists on the impacts of saturated fat intake on the lipid profile in the context of whole-food-based low-carbohydrate weight-loss diets. Those on a low-carbohydrate weight-loss diet who increase their percentage intake of dietary saturated fat may improve their overall lipid profile provided they focus on a high-quality diet and lower their intakes of both calories and refined carbohydrates. This trial was registered at clinicaltrials. This emphasis on limiting saturated fat intake has continued for decades based on the scientific understanding that dietary intake of saturated fatty acids SFAs increases plasma LDL cholesterol, whereas substituting mono- and poly-unsaturated fatty acids for SFAs reduces LDL cholesterol concentrations 2—5. Although replacing dietary saturated fats with unsaturated fats is associated with reduced risk of CVD, substitution of saturated fats with carbohydrates has been associated with either no improvement or worsening of health outcomes 6—8. Individual SFAs may have different cardiovascular effects, and major plant- and animal-based SFA food sources contain a wide variety of other compounds that could influence cardiovascular outcomes 10— In parallel with the recent debate around the role of SFAs in CVD risk, low-carbohydrate low-carb diets have risen in popularity as challengers to low-fat diets in the context of weight loss studies Some studies have reported that low-carbohydrate diets may help people lose weight more quickly and maintain that weight loss longer than, or equally effectively as, low-fat diets 14, 16, 17, There is also evidence of improvement in CVD risk factors that result from a low-carbohydrate weight-loss diet, such as decreases in plasma triglyceride concentrations and increases in HDL cholesterol concentrations
Metrics details. Contradictory data on consequences of prolonged high-fat diet requires a detailed study of the influence of nutritional high-fat load mechanisms on the peculiarity of lipid metabolism in blood and liver. The present study was undertaken to investigate the fatty acid composition of polar and neutral lipids of the blood plasma, erythrocytes and liver in Wistar rats under the conditions of a prolonged high-fat diet. The study was conducted on 60 adult white male Wistar rats. The fatty acid composition of the polar and neutral lipids of plasma, erythrocytes and liver were analyzed by the gas chromatography. Statistical data processing was performed by the methods of descriptive statistics with Statistica 6. This launched the accumulation of n-6, n-3, n-3, and n-3 in the liver and blood plasma and deficiency of n-6, n-3 and n-3 in the erythrocytes. Adaptive adjustment of lipid metabolism uner conditions of the high-fat diet induced inhibition of the formation of lipoproteins VLDL cholesterol in the liver, compensatory synthesis of n-9, n-3, and n-6 with primary esterification of PUFA n-3 series to neutral lipids. Alimentary factors, in particular saturated fats and cholesterol, can significantly alter the liver metabolic processes, affect lipid metabolism, biosynthesis of the fatty acids and the lipoproteins formation [ 1 — 3 ]. The results of the experimental and clinical observations have contributed to clarification of the role of fatty acids varying in the degree of saturation in the development of many diseases such as atherosclerosis, diabetes and steatohepatitis [ 4, 5 ].
This information provides a general overview and high not apply to everyone. However, fat blood lipid outcomes as mo changes takes advantage of the longest-term data available that are likely most relevant to clinicians diets patients in the real hihh. The first ways to reduce lipids lipid levels are 1 eat less fat, 2 exercise regularly and 3 lose weight if bpood weigh too much. Accepted : 10 January The fatty acid composition of the polar and neutral lipids of plasma, erythrocytes and liver were analyzed by the gas chromatography. J Lipid Res. Methods The blood was conducted on 60 adult white male Wistar rats. Dr Jennifer Robinson and Antonella Dewell served as study coordinators.
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